Quote:"The Longest Living Humans Have High Cholesterol...The Sweden Study Found Those Who Reach The Age Of 100 All Had High Cholesterol."
Dr Ben Bikman, PhD

"Cholesterol Is A Molecule Of Life & LDL Is The Unsung Hero Of The Immune System."

"High Cholesterol Leads To A Longer Life."

The longitudinal Swedish AMORIS cohort study—which has a 35-year follow-up period—has shown that high total cholesterol is linked with reaching 100 years of age.

The decades long study on 812,073 participants showed that high levels of total cholesterol & iron, with low levels of glucose were linked to becoming a centenarian.

Dr Uffe Ravnskov, MD PhD & Dr Malcolm Kendrick found the same evidence, in their numerous publications on cholesterol & longevity. The research showed that those with the highest LDL-C, lived the longest.

LDL is a transport molecule for cholesterol & a vital immune system regulator. Lowering LDL levels with medications drugs does not lengthen life...it shortens it.

For instance, documentation shows that when LDL drops below 70 mg/dL, hemorrhagic strokes (brain bleeds) increase at 3X the rate.

When cholesterol is lowered below 180 mg/dL with medication, the rate of cancer & dementia increases drastically.

Total cholesterol that stays below 150 mg/dL, chronic illnesses like cancer & autoimmune diseases develop.


Every Cell Must Have Cholesterol In Order To Perform These Bodily Functions:
Building block of all cells.
Creation of mitochondria.       
Absorption of Vitamins A, D, E & K.
Critical for digestion & bile acids.
Immune system protection against infections.     
Protection against bacteria, pathogens & viruses.       
Protect against cancer.
Prevent depression & suicide.
Protect against chemicals, toxins & heavy metals.   
Prevent dementia & cognitive decline.
Protect against all cause mortality.
Production of all steroid hormones.
Protect bone density & prevent osteoporosis.       
Protect against stroke & heart disease.
Important for lungs & airways.


The Way Of Life To Keep Glucose Low & Cholesterol High:
A low carbohydrate diet prioritizing nutrient dense animal foods, eliminating harmful seed oils, sugar & processed foods provides the best healthy cholesterol profile.

This approach keeps Triglycerides low & HDL high, which is one of the best Biomarkers for Cardiovascular health. TG/HDL ratio of less than 1.5 is optimal.

Quote:INJECTING CRUD, EJECTING GOD, AND BEING A PATENT SLAVE

May 27, 2024 / Joseph P. Farrell

Every now and then I get a very unusual article from all of you that I have to pass along, together with my usual high octane speculation, and that's the case with today's blog article that was spotted and shared by S.D. This one clearly falls into the high octane questions and speculations  category, and I think you'll see why as soon as you read the title:

Destroying Our Connection to God with Gene Editing Injections

There's nothing for it but to dive right in; from the beginning of the article:

Quote:More than a hundred years ago, Rudolf Steiner wrote that. “In the future, we will eliminate the soul with medicine. Under the pretext of a ‘healthy point of view’, there will be a vaccine by which the human body will be treated... so that the human being cannot develop the thought of the existence of soul and Spirit… He would be extremely smart, but he would not develop a conscience, and that is the true goal of some materialistic circles.”

Most regular readers of this website or participants in our members' vidchats know that I am not a big Rudolf Steiner fan. To put all my cards on the table, I find his writing dry, one dimensional, and unreadable, and the endless claims and assertions without the slightest attempt to cite any source from which he draws some of those assertions to be downright irritating and frustration almost without parallel. After all, he's German, and when it comes to footnotes, he should know better.  But in any case, Steiner's speculation here is one of the few that I think is highly intriguing, given its relationship to what follows in this article, and yes, while I have blogged about this subject before, it's time for a revision and extension of remarks. Consider the relation between Steiner's speculation, and the following exchange from the article on whether or not a  "God gene briefing" at the US Department of Defense was fake news or not:

Quote:A viral video which claims to be a leaked 2005 presentation to the DOD by American geneticist, Dean Hamer, shows a man briefing a room of men about the VMAT2 gene, the God gene, and how it can be suppressed with the use of vaccines.

"Excuse me, on the left over here, we have individuals who are religious fundamentalists, religious fanatics. And this is the expression RTPCR, real time PCR, expression of the VMAT2 gene. Our hypothesis is that these are fanatical people, that they have overexpression of the VMAT2 gene and that by vaccinating against this, we'll eliminate this behavior."

Now this last quotation, in the article, is followed by a little parenthetical comment that alleges it is fake, presumably because the alleged briefing was not a Department of Defense briefing, but a "home movie" that it was just a short film:

Quote:In 2021, former NEWS anchor, Ryan Harper, claimed in the San Francisco Chronicle that he created this video as a short film. But has provided no clear evidence to substantiate this claim. And if it is a hoax, then it appears to be disinformation, False information deliberately spread to employ strategic deceptions to advance political, military, or commercial goals. Because in 2009, Dean Hamer lectured at Marlboro College about this same subject.

"Well, it turns out at least one gene that's involved in spirituality has not been identified. It's one of many different players. The next slide shows it. It's something called the Vesicular Monoamine Transporter number two."

So the "God gene" is real. And apparently so is the effort to deploy means for its attenuation or even complete eradication, for the article goes on to link this article:

Erasing Humanity: Evidence of VMAT2 Deletion

In the second article, a connection is drawn to the covid injections and to the phenomenon that some of its recipients have reported a loss of emotionality, listlessness, of "connection" to others and even themselves. Returning to the first article, we note the following:

Quote:The VMAT2 gene is a protein that carries several vital neurotransmitters to synapses in the brain. It basically controls the central nervous system. Deleting it, known as a knockout, would greatly reduce these neurotransmitters creating an endless list of chronic health issues.

VMAT2 deletion is accomplished using the “SLC18A2 cDNA ORF clone” where “cDNA” is “Complimentary DNA.” Which is used in human cloning and genetically modifying an organism. In 2013, Supreme Court judges ruled that cDNA added to target cells in the human genome, makes a person patent eligible. And there are patents for using this to genetically edit the human VMAT2 gene and delete it. This can easily be done using simple CRISPR technology and is even sold online.

In a 2020 study on mice, VMAT2 gene deletion caused schizophrenia. In a 2022 study it caused Parkinson’s disease. In the American Journal of Human Genetics, a study on deleting the VMAT2 induces an “Intellectual Disability Syndrome” in humans. It causes autoimmune disorders, cerebral palsy, type 1 diabetes, and several other illnesses. Begging the question, as Dr. Love asks, “How long have pharma and the government been reducing our humanity through VMAT2 knockouts?”

VMAT2 deletion also causes fearfulness, psychiatric disorders, cardiac arrhythmia, cancer, and accelerated aging. While it’s unlikely that these sadistic mad scientists can cut us off from God, they are definitely doing their best to murder us. (Emphases added)

That list would seem, at least in part, to record some types of adverse reactions associated with the covid injections. At best, it's suggesting that its attenuation or utter removal results in a loss of reasoning ability, and even of physical control of one's body (both the voluntary and involuntary). But you will note that one of my own previous high octane speculations has been confirmed, for note that the Supreme Court of the USA has already ruled that genetic modifications done to edit out this gene  are patentable...

...in other words, one could envision the Mon(ster)santo-GMO scenario on steroids, whereby "infection" with a modified gene of this nature could lead to assertions of property rights over an individual...
...to a kind of "zombie slavery"...

That of course, leads me to my high octane speculation of the day, which must be asked in the form of a question: one assumes that the "God gene" is not the only such "spirituality-influencing" gene in the human genome, and that there may be other genes and even epigenetic factors involved. The question then becomes, is there or are there present, within animals and particularly the higher beasts, similar genes that enable their domestication, as these genes appear to enable human reason and emotional life?

I suspect there are, and I also suspect that just as "they" have already talked about putting their quackcines into the food supply to be ingested by the human population, they might attempt to do the same to the animals...

...if they haven't already. After all, they've pretty well tainted most of the rest of the food supply, and we do not need to catalogue how: chemtrails and aerial spraying, pesticides, steroids, and of course, genetic modifications (and who knows how many of these additives there really are?).

I'm reminded once again of that marvellous chapter, "Banquet at Belbury" of C.S. Lewis' That Hideous Strength, as these insane, godless, irrational and power-mad scientismists continue their mad experiments, that the animals, at least, have to good sense to revolt. After all, when one tries to remove that logos spermatikos of the "god gene" from humanity one ends up with all sorts of health problems, including mental health problems. One can only imagine what happens when one removes the logoi spermatikoi from the rest of the living world as well.

Quote:Researchers have reported that the SARS-CoV-2 mRNA-LNP vaccine may pose the risk of development and progression of cancer [25-28]. In addition, several case reports have described cancer developing or worsening after vaccination and discussed possible causal links between cancer and mRNA-LNP vaccination [29-34].



Based on the molecular weight of BNT162b2 mRNA (Pfizer-BioNTech), the mRNA content per dose is estimated at 13 trillion molecules and 40 trillion molecules in mRNA-1273 (Moderna) [35,36]. The total number of cells in humans is estimated to be 37.2 trillion [37], making the number of mRNA-LNPs very high, ranging from one-third to the equivalent of the total cell number. After inoculation, the mRNA-LNPs are delivered to various organs, especially the liver, spleen, adrenal gland, ovary, and bone marrow [38]. In one study, vaccine mRNA was detected in the lymph nodes of persons vaccinated with hybridization of a SARS-CoV-2 mRNA vaccine-specific probe 7 to 60 days after the second mRNA-1273 or BNT162b2 dose [39]. Modified mRNA with N1-methyl-pseudouridine could translate a large amount of SARS-CoV-2 spike protein (S-protein) [40]. S-protein emerged on the surface of exosomes in the blood of the vaccinated [41]. Fragments of vaccine-specific recombinant S-protein were found in blood specimens of 50% of vaccine recipients and were still detected three to six months later [42].


A study of more than 50,000 employees at a medical institution in the United States observed the incidence of the Omicron variant epidemic based on the number of vaccine doses received (0, 1, 2, 3, and 4 or more doses) over a period of 26 weeks and showed that the number of vaccines received was positively correlated with the cumulative incidence rate of COVID-19 [46]. Susceptibility to COVID-19 infection after multiple vaccinations may be enhanced by antibody-dependent enhancement [47], immune imprinting [39,48], and immunosuppression [25-27]. This can result in a risk of exposure to viral S-protein in addition to vaccine S-protein for the multiple-vaccinated. These data suggest a significant impact on vaccine recipients, including the large number of mRNA-LNPs that are injected, their rapid and widespread distribution particularly into specific organs, the amount of S-protein produced, its long persistence in the body, and increased susceptibility to infection. Next, we consider each factor that may contribute to the involvement of the mRNA-LNP SARS-CoV-2 vaccine in increasing mortality from all cancers and some specific cancer types.


The SARS-CoV-2 vaccine has been shown to cause immunosuppression and lead to the reactivation of latent viruses such as varicella-zoster virus (VZV, human herpesvirus 3; HHV3) or human herpesvirus 8 (HHV8) in some cases [75,76]. HHV8 is considered oncogenic and can cause Kaposi's sarcoma. Oropharyngeal cancer is reported to be caused by the Ebstein-Barr virus (EBV, HHV4) or human papillomavirus (HPV) [77], which also may be reactivated by possible immunosuppression resulting from vaccination. These phenomena could also help explain the excess deaths from lip/oral/pharyngeal cancer in 2022 when mass vaccination with third and later doses was underway.

Cancer development by SARS-CoV-2 mRNA vaccine
In our study, the AMRs of ovarian cancer, leukemia, prostate, lip/oral/pharyngeal, pancreatic, and breast cancers increased significantly beyond the predicted rates, especially in 2022. All of these cancers are known as estrogen and estrogen receptor alpha (ERα)-sensitive cancers [78-83]. Recent research by Solis et al. on the binding ability of S-protein of SARS-CoV-2 against over 9,000 human proteins has shown that S-protein specifically binds to ERα and upregulates the transcriptional activity of ERα. The addition of estradiol (E2) to human breast cancer cells causes proliferation of the cancer cells, whereas the addition of raloxifene, a selective ERα modulator, inhibits proliferation. Breast cancer cells grow when S is added instead of E2, and the addition of raloxifene inhibits their growth. Solis et al. also mentioned that the finding of S-ERα cytosolic colocalization may lead to a potentiation of membrane-bound ERα signaling [84]. Membrane-bound ERα has been implicated in many pathways, including the activation of c-Myc, which promotes the cell cycle and impacts cancer development [85].


ERα-mediated transcription may induce endogenous DNA double-strand breaks (DSBs) in ER-sensitive cancers [86]. Studies have shown that transcriptionally activated ERα induces DSBs by topoisomerase II and the recently known R-loop/G-quadruplex structures formation, significantly increasing the need for BRCA1 for their repair in breast cancer cells [87-89]. One study showed nuclear translocation of mRNA and S protein with the nuclear localization signal [90], and an in silico bioinformatic analysis showed interactions between the S2 subunit of S-protein and BRCA1, BRCA2, and P53 [91], possibly resulting in their sequestration and dysfunction. The possible co-occurrence of high BRCA1 demand to repair DNA damage caused by activated transcription via ERα bound with S-protein along with dysfunction of BRCA1 sequestrated by S-protein raises concerns about increased cancer risk in ERα-sensitive cells in mRNA-LNP SARS-CoV-2 vaccine recipients.


As mentioned above, there is also great concern about the risk of dysfunction in the crucial cancer suppressor genes, brca2 and P53, as well as BRCA1, through mechanisms involving downregulation of IRF9 through interference by specific miRNA in exosomes [26] and the possible sequestration by the S2 subunit of S-protein in the vaccine [91]. Impaired BRCA1 activity is associated with higher risk of breast, uterine, and ovarian cancer in women and prostate cancer in men, as well as moderately higher risk of pancreatic cancer in both men and women [92]. BRCA2-associated cancers include breast and ovarian cancer in women, prostate and breast cancer in men, and acute myeloid leukemia in children [26]. These findings are highly consistent with our results. 


Additional factors that may contribute to the development of cancer are being investigated. Since endogenous ROS causes oxidative DNA damage [93], excessive oxidative stress resulting from the downregulation of ACE2 by S-protein [57] may contribute to cancer development. One study showed that the downregulation of the Mas receptor promotes the metastasis of epithelial ovarian cancer [94]. ACE2 receptor, bound by S-protein after mRNA-LNP vaccination, may directly cause downregulation and subsequent dysfunction of the Mas receptor, possibly leading to an increased risk of metastasis in vaccinated women with ovarian cancer. The observation that injected LNPs accumulate particularly in the ovaries and bone marrow [38] could better explain our findings of excess mortalities from ovarian cancer and leukemia in 2022. According to an analysis of scientific literature about sex hormone receptors in head and neck squamous cell carcinoma (HNSCC), ERα plays various roles in the biopathology of HNSCC, particularly oropharyngeal cancer. These include promoting DNA hypermutation, facilitating HPV integration, and cooperating with epithelial growth factor receptor (EGFR) [82]. This may explain the increased mortality of lip/oral/pharyngeal cancer in our study.


A recent study showed that SARS-CoV-2 RNA could be reverse-transcribed to DNA and integrated into the human cell genome in vitro [95]. Another study reported that transfected mRNA in the human cells exposed to BNT162b2 leads to unsilencing of the endogenous retrotransposon long interspersed element-1 (LINE-1) and reverse transcription of vaccine mRNA sequences to DNA in the nucleus [96]. Accumulation of vaccine mRNA and reverse-transcribed DNA molecules in the cytoplasm could be expected to induce chronic autoinflammation, autoimmunity, DNA damage, and cancer risk in susceptible individuals [97].


The U.S. Food and Drug Administration (FDA) states in its guidance for the production of viral vaccines for infectious disease, "There are several potential mechanisms by which residual DNA could be oncogenic, including the integration and expression of encoded oncogenes or insertional mutagenesis following DNA integration" [98]. The FDA's guidelines are essential for Japan because Japan's special emergency use authorization depended on FDA approval during the COVID-19 pandemic [99]. Recently, some researchers have reported that several lots of Pfizer-BioNTech and Moderna vaccines contain a certain amount of double-stranded DNA fragments from residual plasmid vectors [100,101]. Some of them mentioned that the amount of residual DNA exceeds the regulatory limits for residual DNA set by the FDA. Given these reports and the FDA's regulatory statement, further investigation is required to determine whether the observed excess cancer deaths following mass vaccination were linked to reported residual DNA in the vaccine.

Conclusions

Statistically significant increases in age-adjusted mortality rates of all cancer and some specific types of cancer, namely, ovarian cancer, leukemia, prostate, lip/oral/pharyngeal, pancreatic, and breast cancers, were observed in 2022 after two-thirds of the Japanese population had received the third or later dose of SARS-CoV-2 mRNA-LNP vaccine. These particularly marked increases in mortality rates of these ERα-sensitive cancers may be attributable to several mechanisms of the mRNA-LNP vaccination rather than COVID-19 infection itself or reduced cancer care due to the lockdown. The significance of this possibility warrants further studies. This article was previously posted to the Zenodo repository server on September 18, 2023.

Quote:A goat kid in Stevens County, Minnesota, has tested positive for highly pathogenic avian influenza (HPAI), marking the first case in the United States of bird flu in a domestic ruminant.



The Minnesota Board of Animal Health announced Wednesday the juvenile goat residing on a farm with HPAI-positive poultry also tested positive for the same virus. This is the first U.S. detection in cattle, sheep, goat or their relatives.





"This finding is significant because, while the spring migration is definitely a higher risk transmission period for poultry, it highlights the possibility of the virus infecting other animals on farms with multiple species," State Veterinarian Dr. Brian Hoefs said. "Thankfully, research to-date has shown mammals appear to be dead-end hosts, which means they’re unlikely to spread HPAI further."





The Board of Animal Health says all the poultry on the farm were already quarantined after HPAI was detected in February. Following the confirmation of HPAI in the goat, the Board of Animal Health quarantined all other animals at the farm.




The Board of Animal Health is working with the U.S. Department of Agriculture to investigate the virus' transmission.


Health officials note the risk to the public is "extremely low" and any risk of infection is limited to those who have direct contact with infected animals. No humans in the U.S. have gotten sick after having contact with mammals infected with bird flu, the Board of Animal Health says.



The Minnesota Department of Health has recommendations for personal protective equipment and is monitoring the health of those who have had direct contact with the infected goats.


Anyone who develops respiratory or gastrointestinal symptoms after exposure to the goats may be voluntarily tested for avian influenza and other respiratory pathogens, the release said.

Quote:Cancer Is Striking More Young People, and Doctors Are Alarmed and Baffled

Cancer is hitting more young people in the U.S. and around the globe, baffling doctors. Diagnosis rates in the U.S. rose in 2019 to 107.8 cases per 100,000 people under 50, up 12.8% from 95.6 in 2000, federal data show. A study in BMJ Oncology last year reported a sharp global rise in cancers in people under 50, with the highest rates in North America, Australia and Western Europe.

Doctors are racing to figure out what is making them sick, and how to identify young people who are at high risk. They suspect that changes in the way we live—less physical activity, more ultra-processed foods, new toxins—have raised the risk for younger generations.

...
The U.S. cancer death rate has dropped by one-third since 1991, thanks to a plunge in smoking and better treatment. Screening to catch cancers earlier, including breast cancer, has helped, too.

Although cancer still strikes older people far more often than the young, the rise in early-onset cancers threatens to stall progress. One in five new colorectal cancer patients in 2019 was under 55, a near doubling since 1995. These younger patients are often diagnosed at late stages. Colorectal cancer death rates among patients over 65 are going down, but for those under 50 they are going up.

“We are seeing more and more young people who don’t fit the classic teaching that cancer is a disease of aging,” said Dr. Monique Gary, medical director of the cancer program at Grand View Health in Pennsylvania.

...

The risk of developing some cancers at a young age has increased for each generation born since the 1950s, studies suggest. One found that people born in the 1990s are at double the risk for early-onset colon cancer and four times the risk for rectal cancer, compared with people born around 1950.

...

Researchers are scrutinizing possible causes ranging from inactive lifestyles to microplastics. Oncologists have found a greater risk of developing colorectal cancer at a young age among women who spent a lot of time sitting in front of the TV. Drinking sugary drinks in high school correlated with higher risk, too. Even being born via caesarean section seemed to link another group of women with higher risk of getting colorectal cancer early.

Deep-fried and highly processed foods have been implicated in other studies of early onset colorectal cancer, while diets with fiber, fruits and vegetables likely lower risks. Cancers including colorectal, breast and pancreatic have been tied to obesity, and studies support a link between excess weight and some early onset cancers.

But doctors said obesity and lifestyle can’t fully account for the plight of the people arriving at their clinics.

...

Some doctors suspect that cancer-causing exposures might have started during patients’ childhoods, something that is difficult to trace. Unlike when smoking drove up lung cancer deaths in the 20th century, doctors suspect there isn’t a single carcinogen responsible for the current trends. Some worry young people’s rising cancer risks are a sign of deeper trouble.

...

Other cancer centers are tracking groups of patients over time. Doctors at Memorial Sloan Kettering are collecting tumor samples and asking young patients about exposure to possible risks, from alcohol to anxiety medication. They are probing patients’ origins, too: How old were your parents when you were born? Were you breast-fed?

Quote:Global trends in incidence, death, burden and risk factors of early-onset cancer from 1990 to 2019

Abstract

Objective This study aimed to explore the global burden of early-onset cancer based on the Global Burden of Disease (GBD) 2019 study for 29 cancers worldwid.

Methods and analysis Incidence, deaths, disability-adjusted life years (DALYs) and risk factors for 29 early-onset cancer groups were obtained from GBD.

Results Global incidence of early-onset cancer increased by 79.1% and the number of early-onset cancer deaths increased by 27.7% between 1990 and 2019. Early-onset breast, tracheal, bronchus and lung, stomach and colorectal cancers showed the highest mortality and DALYs in 2019. Globally, the incidence rates of early-onset nasopharyngeal and prostate cancer showed the fastest increasing trend, whereas early-onset liver cancer showed the sharpest decrease. Early-onset colorectal cancers had high DALYs within the top five ranking for both men and women. High-middle and middle Sociodemographic Index (SDI) regions had the highest burden of early-onset cancer. The morbidity of early-onset cancer increased with the SDI, and the mortality rate decreased considerably when SDI increased from 0.7 to 1. The projections indicated that the global number of incidence and deaths of early-onset cancer would increase by 31% and 21% in 2030, respectively. Dietary risk factors (diet high in red meat, low in fruits, high in sodium and low in milk, etc), alcohol consumption and tobacco use are the main risk factors underlying early-onset cancers.

Conclusion Early-onset cancer morbidity continues to increase worldwide with notable variances in mortality and DALYs between areas, countries, sex and cancer types. Encouraging a healthy lifestyle could reduce early-onset cancer disease burden.

Introduction

Globally, cancer is a significant cause of morbidity and mortality, resulting in a large disease burden.1 According to Global Cancer Statistics 2020, breast cancer with the largest number of 2.3 million new cases accounted for 11.7% of all cancers, followed by lung cancer (11.4%), colorectal cancer (CRC) (10.0 %), while lung cancer was the main cause of cancer death (1.8 million deaths, 18%), followed by CRC (9.4%), liver (8.3%) cancer.1 Cancer is generally more prevalent in adults over 50 years, but the incidence of early-onset cancer (<50 years) has increased worldwide.2 In comparison to later-onset cancer, the increase of early-onset cancer has significant personal and societal ramifications. Moreover, early-onset cancer and the adverse impacts of some corresponding cancer treatments may result in additional health issues during subsequent life cycle,3 which would considerably increase the disease burden associated with early-onset cancers.

...

Quote:The ability to efficiently deliver mRNA to the lung would have applications for vaccine development, gene therapy, and more. Here, Suberi et al. showed that such mRNA delivery can be accomplished by encapsulating mRNAs of interest within optimized poly(amine-co-ester) polyplexes. Polyplex-delivered mRNAs were efficiently translated into protein in the lungs of mice with limited evidence of toxicity. This platform was successfully applied as an intranasal SARS-CoV-2 vaccine, eliciting robust immune responses that conferred protection against subsequent viral challenge. These results highlight the potential of this delivery system for vaccine applications and beyond. 

Quote:Yale University researchers have developed a new airborne method for delivering mRNA right to your lungs. The team has also used the method to vaccinate mice intranasally, opening the door for human testing in the near future.

While scientists are hailing the creation as an easy way to vaccinate the masses, critics wonder if the development of an airborne vaccine could be used for nefarious purposes, including covert bioenhancements, which have already been recommended in academic literature.

By releasing the vaccine in the air, there’s no need to inject each person individually – which is not only time-consuming but difficult if an individual objects to the shot. This isn’t the case with an airborne vaccine, which can be released into the air without consent or even the public’s knowledge.

A similar strategy is being used with mRNA in shrimp, which are too small and numerous to be injected individually. Instead, an oral “nanovaccine” was created to stop the spread of a virus. Shai Ufaz, chief executive officer of ViAqua, which developed the technology, stated:

Quote:Oral delivery is the holy grail of aquaculture health development due to both the impossibility of vaccinating individual shrimp and its ability to substantially bring down the operational costs of disease management while improving outcomes…

[b]Bioethics study promotes covert, compulsory bioenhancement

[/b]

Adding to the story is academic endorsement of the use of compulsory, covert bioenhancements. Writing in the journal [i]Bioethics[/i], Parker Crutchfield with Western Michigan University, Homer Stryker M.D. School of Medicine, discusses moral bioenhancements, which refers to the use of biomedical means to trigger moral improvements.
Drug treatments, including vaccines, and genetic engineering are potential examples of bioenhancements. Further, according to Crutchfield:

Quote:It is necessary to morally bioenhance the population in order to prevent ultimate harm. Moral bioenhancement is the potential practice of influencing a person’s moral behavior by way of biological intervention upon their moral attitudes, motivations, or dispositions.
The technology that may permit moral bioenhancement is on the scale between nonexistent and nascent, but common examples of potential interventions include infusing water supplies with pharmaceuticals that enhance empathy or altruism or otherwise intervening on a person’s emotions or motivations, in an attempt to influence the person’s moral behavior.

Some argue that moral bioenhancements should be compulsory for the greater good. Crutchfield believes this doesn’t go far enough. He also wants them to be covert:

Quote:I take this argument one step further, arguing that if moral bioenhancement ought to be compulsory, then its administration ought to be covert rather than overt. This is to say that it is morally preferable for compulsory moral bioenhancement to be administered without the recipients knowing that they are receiving the enhancement.

He even goes so far as to suggest “a covert compulsory program promotes values such as liberty, utility, equality and autonomy better than an overt program does.” So here we have evidence of academic support for covertly releasing drugs and other bioenhancements onto the public. This, combined with the creation of an airborne mRNA vaccine and the government’s history of experimenting on the public, paints an unsettling picture of the future.

[/url]
The Essential Guide to Lung Cancer: Symptoms, Causes, Treatments, and Natural Approaches
Primary lung cancer, which begins in the lungs, is the leading cause of cancer-related deaths worldwide.
Primary lung cancer, which begins in the lungs, is the leading cause of cancer-related deaths worldwide. (Illustrations from Shutterstock/Designed by The Epoch Times)

By [url=https://www.theepochtimes.com/author/mercura-wang]Mercura Wang

• Adenocarcinoma: Typically, this subtype originates within glandular cells situated on the lung’s outer periphery. It accounts for 40 percent of all lung cancers.
  
• Squamous cell carcinoma: Also known as epidermoid carcinoma, this subtype may initiate in flat, delicate cells known as squamous cells, which form the lining of bronchi, the air passages that extend from the trachea (windpipe) into the lungs. This subtype has a more robust association with smoking than other NSCLC types, accounting for 25 percent of all lung cancers.
  
• Large cell carcinoma: This subtype is an undifferentiated malignant tumor that does not exhibit the distinctive cytological traits of squamous cell carcinoma, adenocarcinoma, or small cell carcinoma. Typically exhibiting a faster growth rate than other non-small cell cancers, it can appear in any part of the lungs and accounts for 10 percent of all lung cancers.
  

• Small cell carcinoma, also known as oat cell cancer.
  
• Combined small cell carcinoma, a mixed tumor involving squamous or glandular cells.
  

• Lung carcinoid tumors, which start in the neuroendocrine cells in the lungs and typically grow slowly.
  
• Other lung tumors, which are tumors that include adenoid cystic carcinomas (from salivary gland-like cells in the trachea or bronchi), lymphomas, sarcomas, and benign lung tumors (e.g., hamartomas).
  

• Worsening or persistent cough (lasting over three weeks).
  
• Breathlessness.
  
• Persistent chest pain that intensifies with deep breaths or coughing.
  
• Persistent shoulder pain.
  
• Wheezing.
  
• Unexplained weight loss.
  
• Constant fatigue.
  
• Trouble swallowing.
  
• Enlarged lymph nodes in the neck, above the collarbone.
  
• Presence of blood in coughed-up mucus.
  
• Headaches.
  
• Night sweats.
  
• Recurrent chest infections.
  
• Alterations in voice, such as hoarseness.
  
• Finger and/or toe clubbing.
  
• Superior vena cava syndrome (swelling of the face and neck from the tumor pressing on a large vein in the chest).
  
• Spinal cord compression: Bladder or bowel control disturbances and/or limb weakness may appear from the tumor pressing on the spinal cord.
  

• Syndrome of inappropriate anti-diuretic hormone (SIADH): If the cancer cells make anti-diuretic hormone (ADH), the kidneys retain water, resulting in reduced salt levels within the bloodstream. Major symptoms include muscle weakness or cramping, loss of appetite, restlessness, confusion, fatigue, nausea, and vomiting. It can cause seizures and comas in severe cases.
  
• Cushing syndrome: When cancer cells produce adrenocorticotropic hormone (ACTH), the body produces excessive amounts of cortisol. Symptoms include elevated blood pressure and blood sugar, a rounded facial appearance combined with thin, weak limbs. It also leads to weight gain in the abdomen, resulting in the development of purple marks there.
  
• Lambert-Eaton syndrome: This is thought to result from the tumor-inducing antibodies against the communication between muscle and nerve. Symptoms include muscular weakness, diminished mobility, and challenges in activities such as getting up from a sitting position, climbing stairs, or lifting objects.
  
• Hypercalcemia: This condition occurs when there are elevated calcium levels in the blood, resulting in frequent urination, increased thirst, constipation, fatigue, dizziness, and cognitive confusion.
  
• Blood clots.
  

• Asbestos: Inhalation of these naturally occurring fibers can lead to their entrapment within the lungs. Asbestos has been used in building materials (e.g., roofing shingles and asbestos cement products), friction products (e.g., brake and transmission parts), and heat-resistant fabrics.
  
• Cadmium: Cadmium, a natural element present in small quantities, is mainly obtained in the United States as a byproduct of producing metals like zinc, lead, and copper. As it is resistant to corrosion, cadmium has been utilized in making batteries, pigments, metal coatings, and plastics. Cadmium is also present in tobacco smoke.
  
• Arsenic: Arsenic, a naturally existing semi-metal, is extensively dispersed throughout the Earth’s crust. Currently, in the United States, copper chromated arsenic (CCA) is used in industrial applications, while organic arsenic compounds find use mainly as pesticides, primarily on cotton plants. Arsenic can enter drinking water from natural sources, and is present in tobacco smoke.
  
• Chromium: The natural chemical element chromium serves various purposes, including its use in steel production, chrome plating, dye and pigment creation, leather tanning, and wood preservation.
  
• Silica: Quartz, found in sand, gravel, clay, granite, and various rocks, represents the predominant crystalline silica form, which is hazardous to the lungs. Currently, millions of U.S. workers are exposed to respirable crystalline silica in a variety of industries.
  
• Diesel exhaust: Several studies involving workers exposed to diesel exhaust have revealed slight yet noteworthy rises in lung cancer risk.
  
• Polluted air: In a study examining almost half a million people from England, South Korea, and Taiwan, about half of the nonsmokers were found to have a mutation in a gene called EGFR. Researchers found that inhaling tiny air particles called PM 2.5 can increase the chances of getting NSCLC, especially in those with EGFR gene mutations.
  

• T indicates the size and extent of the primary tumor.
  
• N indicates the presence of cancer in nearby lymph nodes.
  
• M indicates if the cancer has metastasized to other body parts.
  

• Stage 0: This is the earliest stage where cancer cells are found only in the top layer of cells lining the air passages and have not invaded deeper into the lung tissue.
  
• Stage 1: The lung cancer remains within the lung.
  
• Stage 2: The lung cancer remains within the lung, but the tumor is larger and more invasive than stage 1.
  
• Stage 3: At this stage, the cancer has typically spread to nearby lymph nodes and may involve more extensive invasion into the surrounding tissues.
  
• Stage 4: This is the most advanced stage, indicating that cancer has metastasized to distant organs or tissues.
  

• Limited stage: The cancer exists in one lung and/or in its nearby lymph nodes on the same side of the chest.
  
• Extensive stage: The cancer has spread extensively within the lung containing the tumor, to the opposite lung, to lymph nodes on the opposite side of the chest, or distant parts of the body. This may also involve cancer spreading to the fluid surrounding the lung. 
  

• Smoking.
  
• Radon exposure.
  
• Exposure to asbestos and other substances: People who work in asbestos mines, the auto industry, shipyards, cement plants, plumbing and heating trades, construction, and painters, carpenters, and electricians are susceptible to exposure.
  
• Polluted air exposure.
  
• High-red meat and/or saturated-fat diet: One review of studies found consuming red meat may increase the likelihood of developing lung cancer, whereas processed and white meats, organ meats, and fish do not appear to contribute to the risk. High consumption of total and saturated fats has been linked to a higher risk of lung cancer, particularly among smokers.
  
• Older age: It is rare for those under 40 to have this type of cancer.
  
• Ethnicity: Black men have a roughly 12 percent higher likelihood of developing lung cancer than white men, whereas the rate is approximately 16 percent lower for black women than for white women. While the overall risk of lung cancer is lower among Asian Americans, Native Hawaiians, and Pacific Islanders, it is this group's primary cause of cancer-related mortality. Among these, Native Hawaiians exhibit the highest lung cancer death rates.
  
• Personal or family history: Individuals who previously had lung cancer face an elevated chance of recurrence. Around 8 percent of lung cancers stem from inheritance or a genetic predisposition, such as chromosome 6.
  
• Radiation exposure: Cancer survivors who underwent chest radiation therapy face an elevated risk of lung cancer, particularly if they smoke. Individuals exposed to ionizing radiation from atomic bomb detonations or nuclear accidents are more likely to have an increased risk.
  
• Certain diseases: Ongoing lung disorders, such as chronic obstructive pulmonary disease (COPD), increase the likelihood of lung cancer. Lung cancer risk also increases for people with certain autoimmune disorders, including lupus, rheumatoid arthritis, Crohn’s disease, Graves’ disease/hyperthyroidism, and sarcoidosis.
  
• Weakened immune system: Although human immunodeficiency virus (HIV) doesn’t cause cancer per se, since it leads to a weakened immune system, people with HIV/acquired immune deficiency syndrome (AIDS) are prone to developing certain types of cancer, including lung. People with a suppressed immune system are also at an increased risk.
  
• Sedative lifestyle: Smokers who engaged in vigorous activity at least once a week have been found to have a 29 percent lower likelihood of developing lung cancer compared to those who rarely or never participated in such activities.
  
• Beta-carotene supplements: Beta carotene is a red-orange pigment found in many plants and an antioxidant that protects the body from damaging molecules called free radicals. However, research has demonstrated a compelling link between beta-carotene supplements and an increased lung cancer risk among smokers. Therefore, smokers are recommended to obtain beta carotene only via natural food sources.
  

• Chest X-ray: A chest X-ray is often the first test used to search for any abnormal areas in your lungs by looking at the organs and bones inside your chest.
  
• CT scan: A computed tomography (CT or CAT) scan is often the second test during the lung cancer diagnosis procedure. This specialized form of X-ray imaging captures detailed images of the lungs. It is more effective in revealing lung tumors compared to standard chest X-rays. Additionally, it can provide information about the dimensions, form, and location of lung tumors and identify enlarged lymph nodes that may harbor metastatic cancer.
  
• PET scan: A positron emission tomography (PET) scan is a type of nuclear medicine imaging examination that employs radioactive sugar to generate vivid 3D color images, revealing the functioning of your cells. This test is helpful when your doctor suspects the cancer has metastasized but is unsure of its location, and it can also be used in lung cancer staging.
  
• MRI scan: A magnetic resonance imaging (MRI) scan provides detailed cross-sectional images of soft tissues in the body but employs radio waves and strong magnets instead of X-rays. It is particularly valuable in searching for potential lung cancer spread to the brain, heart, or spinal cord.
  
• Ultrasound: Ultrasound employs high-frequency sound waves to generate images of body components. It detects fluid accumulation around the lungs and assists in guiding biopsies or the removal of chest cavity fluids.
  
• Sputum cytology: Sputum, the mucus coughed up from the lungs, is examined under a microscope by a pathologist to detect cancer cells. This process may help identify cancers originating in the major lung airways.
  
• Biopsy: A biopsy is a procedure where a small tissue or tumor sample is extracted from the body for microscopic examination. It is performed to diagnose lung cancer or determine if an anomaly is cancerous. A biopsy is the most effective method to determine whether lung cancer exists and the extent to which it has spread.
  

• Blood clots: A great risk of developing deep vein thrombosis (DVT) exists due to the effects of the cancer itself on clotting factors and sometimes due to chemotherapy. In some cases, clots can dislodge and travel to the lungs, resulting in a condition known as pulmonary embolism (PE).
  
• Paraneoplastic syndromes: These are most prevalent among middle-aged to older adults and are frequently associated with lung cancer.
  
• Chemotherapy-induced nausea and vomiting.
  
• Fatigue.
  
• Shortness of breath: Lung tumors can lead to shortness of breath by obstructing airways, thus hampering airflow. Lung cancer might also lead to pleural effusion, causing fluid accumulation around the lungs and hindering their full expansion. Additionally, lung cancer can reduce red blood cells, affecting oxygen transport.
  
• Hemoptysis (coughing up blood): Hemoptysis is when you cough up sputum with blood. This blood can come from your lungs or the tubes that bring air to your lungs.
  
• Cachexia: This is commonly seen in advanced lung cancer patients with associated weight loss, including muscle loss. At diagnosis, 60 percent of patients have noticeably lost weight.
  
• Anemia: Anemia is common among individuals with lung cancer, with its occurrence and intensity influenced by factors such as the stage of the tumor, duration of the illness, and past and ongoing treatments.
  
• Neutropenia: Chemotherapy-induced neutropenia refers to a decreased level of neutrophils, a type of white blood cell.
  
• Nephrotoxicity: Nephrotoxicity is a swift decline in kidney function caused by the toxic impact of medications and chemicals.
  
• Fluid buildup in the chest.
  
• Hypercalcemia: In some instances, lung cancer can cause hypercalcemia, which is higher calcium levels in the blood.
  

• Wedge resection or segmentectomy: This surgery removes a small portion of the lung. It’s only suitable for a select group of patients with small, localized cancers, often at an early stage of NSCLC.
  
• Lobectomy: This involves removing one of the larger sections (lobes) of the lung. It is usually recommended if the cancer is confined to one specific section of one lung.
  
• Pneumonectomy: In this procedure, the entire lung is removed. It’s chosen when the cancer is located in the middle of the lung or has extensively spread throughout the lung.
  
• Sleeve resection: This procedure removes part of the bronchus.
  

• External beam radiation therapy (EBRT): EBRT directs radiation from outside the body onto the cancer. This method is painless, with each session taking just a few minutes. Lung radiation treatments are usually administered five days a week over five to seven weeks.
  
• Internal radiation therapy (brachytherapy): Also known as endobronchial brachytherapy or endobronchial radiation therapy, this is a form of internal radiation therapy. It involves placing radioactive material inside the bronchus during a bronchoscopy. Brachytherapy is utilized for lung tumors in the bronchi and minimizes radiation exposure to nearby organs.
  

• Local hyperthermia: Heat is applied to a small area of the body.
  
• Regional hyperthermia: Heat is applied to an organ, limb, or large area of the body.
  
• Whole-body hyperthermia: A patient is treated in a thermal chamber or covered with heated blankets, elevating the body temperature to 107 or 108 F for brief durations.
  

• Astragalus: Astragalus membranaceus polysaccharides (APS), one component of the JFK prescription, have been found to decrease the proliferation and migration of NSCLC cells.
  
• Berberine: Berberine comes from plants like Chinese medicinal herbs barberry and goldenseal. One study showed that a particular form of berberine called phytantriol-based berberine has about 20 times the strength in stopping lung cancer growth and spread as regular berberine. In lab tests, it also helped control genes related to lung cancer, reducing the activity of proteins that enhance cancer cell growth. It also has fewer side effects than regular berberine.
  
• White mulberry (Morus alba): Albanol B (ABN-B), isolated from mulberries, has been found to be able to inhibit the proliferation of four human lung cancer cell lines and induce cell death. Treatment with Kuwanon M (KWM), another characteristic constituent isolated from the root bark of Morus alba, can induce the suppression of lung cancer cell proliferation and migration in lab studies.
  
• Snake-needle grass (Oldenlandia diffusa): In one animal study, this extract effectively suppressed lung cancer cells and triggered a notable increase in their death. Additionally, it significantly inhibited lung metastases without any noticeable adverse effects.
  
• Curcumin: Curcumin, derived from turmeric, is a key compound with various therapeutic properties, like antioxidants and anti-inflammatory effects. It can be an effective adjunct in treating solid organ tumors due to its ability to regulate oncogenes and its lack of harmful effects. However, most evidence comes from laboratory studies, so more research involving patients is needed to confirm its effectiveness.
  
• Thai herbal extracts: One study found several extracts of Thai medicinal herbs Bridelia ovata, Croton oblongifolius (dyer’s croton), and Erythrophleum succirubrum that exhibited toxic effects on one lung cancer cell line. The ethanolic extract of E. succirubrum was the most effective and had a significant potential to be used for future lung cancer treatment.
  

• Insulin potentiation therapy (IPT): Also known as insulin-potentiated targeted low-dose therapy (IPTLD), this offers a complementary approach to treating certain cancers, including lung cancer. Often termed “low-dose chemotherapy,” IPT is a complementary treatment alongside other cancer therapies. As insulin can increase the permeability of cancer cell membranes due to their higher insulin receptor count, this therapy makes it easier for chemotherapy drugs to enter the cancer cells. Then, fewer chemotherapy chemicals are needed for the same therapeutic effects.
  
• Magnetic aerosol drug targeting (MADT): An emerging technique that shows great promise for lung cancer therapy, MADT involves utilizing a permanent magnet to deliver drugs to specific locations within the lung precisely. MADT can be performed by using either an implanted magnet within the body or a magnet positioned externally. Using a permanent magnet has a noticeable impact on how efficiently treatment particles are trapped.
  

• Avoid or quit smoking or being around secondhand smoke: Within 12 years of quitting, your lung cancer risk will decrease by over half that of a smoker.
  
• Test your home's radon levels: You can get the radon level of your home tested by contacting your state radon office.
  
• Avoid or minimize exposure to carcinogens: Before taking a new job, do your due diligence to determine if there will be exposure to lung cancer-causing toxic substances such as asbestos and silica.
  
• Eat a lower-fat, high-fiber diet: Choose a diet with lots of fruits, vegetables, and healthy meats. Limit red meat and saturated fats; consume poultry, fish, and vegetables instead. Regular consumption of cruciferous vegetables, such as cabbage, broccoli, and Brussels sprouts, has been shown to protect against lung cancer.
  
• Get enough omega-3s: The three primary omega-3 fatty acids include alpha-linolenic acid (ALA) found in plant oils, eicosapentaenoic acid (EPA) found in fish and other seafood, and docosahexaenoic acid (DHA) found in seafood. Research suggests that omega-3 fatty acids might hinder the growth of cancer cells and potentially regulate key pathways that influence the advancement or complications of lung cancer.
  
• Boost your vitamins and antioxidants: Oxidative stress is a key factor in the harmful effects caused by cancer radiation therapy and chemotherapy. Antioxidants such as vitamins A, C, and E, as well as selenium and zinc, can potentially enhance the body’s defense against oxidative stress. On the other hand, laboratory research indicates that very high doses of vitamin C make it act as a pro-oxidant, reducing the energy levels in cancer cells, damaging their DNA, and thus triggering a chain of reactions that eventually leads to their death.
  
• Exercise at least twice per week: Adults should aim for 150 minutes of moderate-intensity aerobic activity per week, coupled with strength training twice a week.
  
• Get lung cancer screenings if at high risk: If you are at a high risk of developing lung cancer, talk with your doctor, who will order the appropriate screening tests for you.
  

Quote:The Centers for Disease Control and Prevention (CDC) recently issued a warning about a bacterial infection that people can get by eating raw or undercooked fish or by exposing an open wound to coastal waters. A friend tells us this San Jose woman likely got this specific infection after eating undercooked tilapia.



A San Jose mother’s life is changed forever. Laura Barajas, 40, has had her limbs amputated while battling a bacterial infection.


“It’s just been really heavy on all of us. It’s terrible. This could’ve happened to any of us,” said Barajas’ friend Anna Messina.


Messina says back in late July Barajas had bought tilapia from a local market for dinner. She cooked it and ate it alone. Within days, she got very ill and was then hospitalized.


“She almost lost her life. She was on a respirator,” Messina said. “They put her into a medically induced coma. Her fingers were black, her feet were black her bottom lip was black. She had complete sepsis and her kidneys were failing.”

Now, a month and a half later, Barajas is without her arms and legs.
Messina believes the infection was caused by Vibrio vulnificus — a bacterial infection the CDC has been warning about.


“The ways you can get infected with this bacteria are one-you can eat something that’s contaminated with it the other way is by having a cut or tattoo exposed to water in which this bug lives,” said UCSF Infectious Disease Expert Dr. Natasha Spottiswoode.


Spottiswoode says the bacteria is especially concerning for people who are immunocompromised.

The CDC says about 150-200 cases of the infections are reported each year and about one in five people with the infection die — sometimes within one to two days of becoming ill.

Quote:Despite current controversies, some reports show a paradoxical mitigating effect associated with smoking in individuals with symptomatic COVID-19 compared to the general population. To explain the potential mechanisms behind the lower number of hospitalized COVID-19 patients, it has been hypothesized that cigarette smoking may reduce the odds of cytokine storm and related severe inflammatory responses through cholinergic-mediated anti-inflammatory mechanisms. Japanese scientists have recently identified a potential mechanism behind the lower numbers of COVID-19 cases amongst smokers compared to non-smokers. However, we believe that this mitigative effect may be due to the relatively high concentration of deposited energy of alpha particles emitted from naturally occurring radionuclides such as Po-210 in cigarette tobacco. Regarding COVID-19, other researchers and our team have previously addressed the anti-inflammatory and immune-modulating effects of low doses of ionizing radiation. MC-simulation using the Geant4 Monte Carlo toolkit shows that the radiation dose absorbed in a spherical cell with a radius of .9 μm for a single 5.5 MeV alpha particle is about 5.1 Gy. This energy deposition may trigger both anti-inflammatory and anti-thrombotic effects which paradoxically lower the risk of hospitalization due to COVID-19 in smokers.

Quote:A team of Japanese scientists demonstrates that treatment with AHR agonists decreases expression of ACE2 via AHR activation, resulting in suppression of COVID-19.


Researchers have identified two drugs that mimic the effect of chemicals in cigarette smoke to bind to a receptor in mammalian cells that inhibits production of ACE2 proteins, a process that appears to reduce the ability of the SARS-CoV-2 virus to enter the cell.

Quote:Results: The percentages of current smokers (4.1% vs 16%, p = .00003) and never smokers (71.6% vs 56.8%, p = .0014) were significantly different between COVID-19 and non-COVID 19 patients. COVID-19 patients had lower mean age (69.5 vs 74.2 years, p = .00085) and were more frequently males (59.2% vs 44%, p = .0011). In the logistic regression analysis, current smokers were significantly less likely to be hospitalized for COVID-19 compared with nonsmokers (odds ratio = 0.23; 95% confidence interval, 0.11-0.48, p < .001), even after adjusting for age and gender (odds ratio = 0.14; 95% confidence interval, 0.06-0.31, p < .001).

Conclusions: We reported an unexpectedly low prevalence of current smokers among COVID-19 patients hospitalized in nonintensive care wards. The meaning of these preliminary findings, which are in line with those currently emerging in literature, is unclear; they need to be confirmed by larger studies.

Implications: An unexpectedly low prevalence of current smokers among patients hospitalized for COVID-19 in some Italian nonintensive care wards is reported. This finding could be a stimulus for the generation of novel hypotheses on individual predisposition and possible strategies for reducing the risk of infection from SARS-CoV-2 and needs to be confirmed by further larger studies designed with adequate methodology.

Quote:President Biden said Friday he plans to request additional funding from Congress for the development of a new COVID-19 vaccine, adding he may require everyone to take it whether they previously received a vaccine or not.

Quote:The Biden administration is buying Covid equipment and hiring pandemic “safety protocol” officers. And the federal government is also sending $1.4 billion to defense contractors and pharmaceutical companies for more Covid “countermeasures” and vaccines. Does the lunacy ever stop? 

The Covid fanatics’ nonsense never seems to end. In 20 years, we will still probably be prepping for our next lockdown and queuing for our 100th booster shot. And why would it end? Biden’s Project NextGen proves that no lessons have been learned from the last set of pandemic restrictions. Even the British government – certainly not one you could accuse of failure to lock down and vaccinate – is doing none of this.

We the people are sick of it, and are starting to resist. Only 17% of Americans got their Covid booster shot according to CDC data. Americans constantly read alarmist stories about new variants and potential future lockdowns, and have started saying “hell no” to the tyranny. 

But it is easy to say things online, and much more difficult to muster the strength to refuse to comply in the face of shame, vitriol, and the admonishment of those around you.

It was hard the first time to fight back against the diktats of those passing their lockdown, mask and vaccine laws, especially when they didn’t even follow their own rules and principles. It was new, and we didn’t know what to do at first. We didn’t know what Covid really was and how it would affect us in the long run. Nobody had all the answers. But now we do. We know that Dr Fauci misled the American people about the potential origin of the Covid virus, quickly dismissing the Wuhan lab leak theory as a “conspiracy”. We know the mask mandates forced upon citizens were ineffective at best. And we know the devastating impact lockdowns have had on the mental health of young people. 

We have but one option. Do not comply. As soon as we give up our freedom for a bit of safety we risk losing all of it, alongside everything that gives us our humanity. I urge everyone reading this today to take into account what we would be risking by sleepwalking again into disaster. 

Our jobs, our friendships, and even our futures may be at risk if we refuse to back down in the face of Biden’s fearmongering. But we must be brave and stand up for our right to a free life. It is the only option we have. 

Quote:A new study finds developed countries requiring the most vaccine doses for infants have higher childhood mortality rates, contradicting assumptions that more vaccines equate to lower deaths. The data suggest unintended consequences may increase all-cause mortality

Quote:Conclusions


There are statistically significant positive correlations between mortality rates of developed nations and the number of early childhood vaccine doses that are routinely given. Further investigations of the hypotheses generated by this study are recommended to confirm that current vaccination schedules are achieving their intended objectives.